Testicular infarction by spermatic cord torsion
Coming in from the cord on top to the testicle, there's a thick-walled artery and there's a thin-walled vein. The classic mechanism by which testicular infarction occurs is that the testicle twists on this cord. When it twists, the artery continues to allow to blood to flow in because it's thick-walled, however the vein collapses, and when the vein collapsed the blood comes in but blood can't go out. This is one way by which an ischemia happens, by blocking of the venous supply and the result is that fresh blood would not be passing across the organ. So, the tissue will die because the occlusion of the vein, and blood continue to enter because the artery remains open relative to the blockage of the vein. As blood piles up within the testicle, the tissue gets relatively loose and that will eventually create a red infarction (hemorrhagic). Unlike a white (anemic) infarction which is wedge-shaped and pale in color based on an anatomical principle of a blockage of a feeding vessel (arterial blood supply) to the organ that limits the amount of nutrients (blood/oxygen/glucose) that can flow into the area of ischaemic necrosis. By: https://www.instagram.com/p/Caho7IWAAoe/?utm_source=ig_web_copy_link
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